Targeting gap junction intercellular communication as a potential therapy for HCV-related carcinogenesis

Author:

Abdulrahman Almatrrouk Shaihana1,Oliver Anthony W1,Hampson Lynne1,Hampson Ian N1

Affiliation:

1. University of Manchester, Viral Oncology, Research Floor, St Mary's Hospital, Manchester, UK

Abstract

ABSTRACT:  Worldwide, at least 170 million people are infected with hepatitis C virus (HCV), which is associated with hepatocellular carcinoma (HCC). With the recent success of Sofosbuvir (and other agents) antiviral therapy may be used as a future early-stage HCC treatment; however, in the short term, a cost-effective solution is needed to treat patients with viral-associated HCC. Here, we emphasize the potential of targeting gap junction intercellular communication (GJIC) as a therapeutic approach for HCC as HCV perturbs GJIC, which is linked to cellular transformation. We review the ROCK inhibitor Y-27632 and structurally related compounds that may inhibit the carcinogenic properties of HCV.

Publisher

Future Medicine Ltd

Subject

Virology

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