Unregulated Insulin Secretion by Pancreatic Beta Cells in Hyperinsulinism/Hyperammonemia Syndrome: Role of Glutamate Dehydrogenase, ATP-Sensitive Potassium Channel, and Nonselective Cation Channel-Reference-Cited by-同舟云学术

Unregulated Insulin Secretion by Pancreatic Beta Cells in Hyperinsulinism/Hyperammonemia Syndrome: Role of Glutamate Dehydrogenase, ATP-Sensitive Potassium Channel, and Nonselective Cation Channel

Published:2006-03 Issue:3 Volume:59 Page:359-364
ISSN:0031-3998
Container-title:Pediatric Research
language:
Short-container-title:Pediatr Res

Author:

Kawajiri Mie,Okano Yoshiyuki,Kuno Miyuki,Tokuhara Daisuke,Hase Yutaka,Inada Hiroshi,Tashiro Fumi,Miyazaki Jun-Ichi,Yamano Tsunekazu

Publisher

Springer Science and Business Media LLC

Subject

Pediatrics, Perinatology and Child Health

Reference30 articles.

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3. Hyperinsulinism and Hyperammonemia in Infants with Regulatory Mutations of the Glutamate Dehydrogenase Gene

4. Molecular basis and characterization of the hyperinsulinism/hyperammonemia syndrome: predominance of mutations in exons 11 and 12 of the glutamate dehydrogenase gene. HI/HA Contributing Investigators.

5. Hyperinsulinism and Hyperammonemia Syndrome: Report of Twelve Unrelated Patients

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