Cortical Consequences of In Vivo Blockade of Monocarboxylate Transport During Brain Development in Mice-Reference-Cited by-同舟云学术

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Cortical Consequences of In Vivo Blockade of Monocarboxylate Transport During Brain Development in Mice

Published:2007-01 Issue:1 Volume:61 Page:54-60
ISSN:0031-3998
Container-title:Pediatric Research
language:
Short-container-title:Pediatr Res

Author:

Adle-Biassette Homa,Olivier Paul,Verney Catherine,Fontaine Romain H,Evrard Phillippe,Hénin Dominique,Massias Laurent,Gressens Pierre,Baud Olivier

Publisher

Springer Science and Business Media LLC

Subject

Pediatrics, Perinatology, and Child Health

Reference30 articles.

1. Expression of monocarboxylate transporter mRNAs in mouse brain: Support for a distinct role of lactate as an energy substrate for the neonatal vs. adult brain

2. Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization.

3. Glucose metabolism in the developing brain

4. Glycogen in astrocytes: possible function as lactate supply for neighboring cells

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1. Proteomic and transcriptomic study of brain microvessels in neonatal and adult mice;PLOS ONE;2017-01-31

2. Major remodeling of brain microvessels during neonatal period in the mouse: A proteomic and transcriptomic study;Journal of Cerebral Blood Flow & Metabolism;2016-07-21

3. Glycolysis-mediated control of blood-brain barrier development and function;The International Journal of Biochemistry & Cell Biology;2015-07

4. Early undernutrition induces glucagon resistance and insulin hypersensitivity in the liver of suckling rats;American Journal of Physiology-Endocrinology and Metabolism;2012-05-01

5. Molecular biology of the blood-brain and the blood-cerebrospinal fluid barriers: similarities and differences;Fluids and Barriers of the CNS;2011-01-18

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