Graves’ Disease Complicated by Pregnancy: Malformation and Thyroid Dysfunction in Fetus Related to Maternal Disease or Its Treatment
Author:
Publisher
Japanese Society for Pediatric Endocrinology
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Pediatrics, Perinatology, and Child Health
Link
https://www.jstage.jst.go.jp/article/cpe/7/2/7_2_73/_pdf
Reference19 articles.
1. Effects of thyroid antagonists on rat embryos cultured in vitro
2. 2) Momotani N, Ito K, Hamada N, et al. Maternal hyperthyroidism and congenital malformation in the offspring. Clin Endocrinol 1984; 20: 695-700.
3. 3) Stanisstreet M, Pharoah POD, Herbert LC. Culture of rat embryos in sera from human hyperthyroid subjects. Med Sci Res 1988; 16: 1105-6.
4. 4) Harakawa S, Akazawa S, Akazawa M, et al. Changes of serum thyroid hormone levels induce malformations on early embryogenesis in rats. Acta Endocrinol (Copenh) 1989; 121: 739-43.
5. 5) Sparre LS. Maternal free thyroxine and thyroxine-binding globulin during pregnancy ending in congenital malformations in the offspring. Gynecol Obstet Invest 1989; 27: 19-21.
Cited by 1 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Transient Central Hypothyroidism Due to Pituitary Suppression in a Premature Neonate Born to a Mother with Three-Year-Untreated Graves' Disease;Clinical Pediatric Endocrinology;2003
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