Author:
Dzeranova L K,Paramonov V M,Rozhinskaya L Ya,Pleshcheva A V,Manchenko O V,Vorontsov A V,Kolesnikova G S,Grigor'ev A Yu,Pigarova E A
Abstract
The reported clinical case represents an example of characteristic traits in lipid metabolism and fat tissue deposition in hypercorticosolism.
Probable causes for fat mass expansion in glucocorticosteroid excess, its distinctive cushingoid distribution, role of
11β-HSD1 and glucocorticoid receptors and reversibility of specific abnormalities after correction of hypercortisolism are discussed in
the context of physiological and pathological aspects of glucocorticosteroid action.
Publisher
Endocrinology Research Centre
Subject
Public Health, Environmental and Occupational Health,Nutrition and Dietetics,Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Cited by
1 articles.
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