Downregulation of TIM-3 mRNA expression in peripheral blood mononuclear cells from patients with systemic lupus erythematosus
Author:
Affiliation:
1. China Medical University, China; China Medical University, China
2. China Medical University, China
3. China Medical University, China; China Medical University, China; China Medical University, China
Publisher
FapUNIFESP (SciELO)
Subject
Cell Biology,General Pharmacology, Toxicology and Pharmaceutics,General Medicine,Immunology,Physiology,Biochemistry,General Neuroscience,Biophysics,General Medicine
Link
http://www.scielo.br/pdf/bjmbr/v48n1/1414-431X-bjmbr-1414-431X20143701.pdf
Reference28 articles.
1. TIM genes: a family of cell surface phosphatidylserine receptors that regulate innate and adaptive immunity;Freeman GJ;Immunol Rev,2010
2. Tim-3 inhibits T helper type 1-mediated auto- and alloimmune responses and promotes immunological tolerance;Sanchez-Fueyo A;Nat Immunol,2003
3. Identification of Tapr (an airway hyperreactivity regulatory locus) and the linked Tim gene family;McIntire JJ;Nat Immunol,2001
4. T Cell Ig- and mucin-domain-containing molecule-3 (TIM-3) and TIM-1 molecules are differentially expressed on human Th1 and Th2 cells and in cerebrospinal fluid-derived mononuclear cells in multiple sclerosis;Khademi M;J Immunol,2004
5. The Tim-3 ligand galectin-9 negatively regulates T helper type 1 immunity;Zhu C;Nat Immunol,2005
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2. Tim-3, PD-1, CD244 and Foxp3 Positive T Cells Relation to the Prognosis of Dermatomyositis and Polymyositis Patients;Journal of the College of Physicians and Surgeons Pakistan;2023-04-01
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5. Tim-1 alleviates lupus nephritis-induced podocyte injury via regulating autophagy;Central European Journal of Immunology;2021
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