Subacute cognitive impairment, hyponatremia and mesial temporal lobe lesions: a typical presentation of voltage-gated potassium channel (VGKC) antibody-associated limbic encephalitis-Reference-Cited by-同舟云学术

Subacute cognitive impairment, hyponatremia and mesial temporal lobe lesions: a typical presentation of voltage-gated potassium channel (VGKC) antibody-associated limbic encephalitis

Published:2011-12 Issue:6 Volume:69 Page:990-991
ISSN:0004-282X
Container-title:Arquivos de Neuro-Psiquiatria
language:
Short-container-title:Arq. Neuro-Psiquiatr.

Author:

Saccardi Thais¹,Pedroso José Luiz¹,Bernardino Tenille¹,Barsottini Orlando G.P.¹,Ferraz Henrique Ballalai¹

Affiliation:

1. Universidade Federal de São Paulo, Brazil

Publisher

FapUNIFESP (SciELO)

Subject

Neurology,Clinical Neurology

Link

http://www.scielo.br/pdf/anp/v69n6/28.pdf

Reference5 articles.

1. Autoimmune limbic encephalitis in 39 patients: immunophenotypes and outcomes;Bataller L;J Neurol Neurosurg Psychiatry,2007

2. Investigation of LGI1 as the antigen in limbic encephalitis previously attributed to potassium channels: a case series;Lai M;Lancet Neurol,2010

3. Neuronal surface antigen antibodies in limbic encephalitis: clinical: immunologic associations;Graus F;Neurology,2008

4. Reversible limbic encephalitis with antibodies against the membranes of neurones of the hyppocampus;Shimazaki H;J Neurol Neurosurg Psychiatry,2007

5. Manegement of voltage-gated potassium channel antibody disorders;Merchut MP;Neurol Clin,2010

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