Myocyte necrosis is the basis for fibrosis in renovascular hypertensive rats

Author:

Okoshi M.P.,Matsubara L.S.,Franco M.1,Cicogna A.C.,Matsubara B.B.

Affiliation:

1. Universidade Estadual Paulista

Publisher

FapUNIFESP (SciELO)

Subject

Cell Biology,General Pharmacology, Toxicology and Pharmaceutics,General Medicine,Immunology,Physiology,Biochemistry,General Neuroscience,Biophysics

Reference38 articles.

1. Myocardial mechanical alterations during gradual onset long-term hypertension in rats;Capasso JM;American Journal of Physiology,1981

2. Papillary muscle structure-function relations in the aging spontaneously hypertensive rat;Bing OHL;Clinical and Experimental Hypertension,1988

3. Contractile function, myosin ATPase activity and isozymes in the hypertrophied pig left ventricle after a chronic progressive pressure overload;Wisenbaugh T;Circulation Research,1983

4. Myocardial, biochemical, contractile, and electrical performance after imposition of hypertension in young and old rats;Capasso JM;Circulation Research,1986

5. Performance of papillary muscles from the aging spontaneously hypertensive rat: temporal changes in isometric contraction parameters;Cohen ME;Proceedings of the Society for Experimental Biology and Medicine,1987

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