Effect of endogenous hydrogen sulfide inhibition on structural and functional renal disturbances induced by gentamicin

Author:

Francescato H.D.C.1,Chierice J.R.A.1,Marin E.C.S.1,Cunha F.Q.1,Costa R.S.1,Silva C.G.A.1,Coimbra T.M.1

Affiliation:

1. Universidade de São Paulo, Brasil

Publisher

FapUNIFESP (SciELO)

Subject

Cell Biology,General Pharmacology, Toxicology and Pharmaceutics,General Medicine,Immunology,Physiology,Biochemistry,General Neuroscience,Biophysics

Reference20 articles.

1. Prevalence and risk factors for aminoglycoside nephrotoxicity in intensive care units;Oliveira JF;Antimicrob Agents Chemother,2009

2. Role of myofibroblasts, macrophages, transforming growth factor-beta endothelin, angiotensin-II, and fibronectin in the progression of tubulointerstitial nephritis induced by gentamicin;Geleilete TJ;J Nephrol,2002

3. Inhibition of nuclear factor-kappaB activation attenuates tubulointerstitial nephritis induced by gentamicin;Volpini RA;Nephron Physiol,2004

4. Role of immunocompetent cells in nonimmune renal diseases;Rodriguez-Iturbe B;Kidney Int,2001

5. Hydrogen sulfide is a novel mediator of lipopolysaccharide-induced inflammation in the mouse;Li L;FASEB J,2005

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