Necrostatin-1 Prevents Necroptosis in Brains after Ischemic Stroke via Inhibition of RIPK1-Mediated RIPK3/MLKL Signaling
Author:
Publisher
Aging and Disease
Subject
Cell Biology,Clinical Neurology,Geriatrics and Gerontology,Pathology and Forensic Medicine
Link
http://www.aginganddisease.org/fileup/2152-5250/PDF/ad-0-0-0-1809041226-1.pdf
Reference34 articles.
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3. Puyal J, Ginet V, Clarke PGH (2013). Multiple interacting cell death mechanisms in the mediation of excitotoxicity and ischemic brain damage: A challenge for neuroprotection. Prog Neurobiol, 105: 24-48.
4. Holler N, Zaru R, Micheau O, Thome M, Attinger A, Valitutti S, et al. (2000). Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule. Nat Immunol, 1: 489-495.
5. Degterev A, Huang Z, Boyce M, Li Y, Jagtap P, Mizushima N, et al. (2005). Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury. Nat Chem Biol, 1: 112-119.
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