Angelica Sinensis Polysaccharide Suppresses Epithelial-Mesenchymal Transition and Pulmonary Fibrosis via a DANCR/AUF-1/FOXO3 Regulatory Axis
Author:
Publisher
Aging and Disease
Subject
Cell Biology,Neurology (clinical),Geriatrics and Gerontology,Pathology and Forensic Medicine
Link
http://www.aginganddisease.org/fileup/2152-5250/PDF/ad-0-0-0-1905131122-1.pdf
Reference44 articles.
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2. Diaz-Pina G, Ordonez-Razo RM, Montes E, Paramo I, Becerril C, Salgado A, et al. (2018). The Role of ADAR1 and ADAR2 in the Regulation of miRNA-21 in Idiopathic Pulmonary Fibrosis. Lung, 196:393-400.
3. Willis BC, Liebler JM, Luby-Phelps K, Nicholson AG, Crandall ED, du Bois RM, et al. (2005). Induction of epithelial-mesenchymal transition in alveolar epithelial cells by transforming growth factor-beta1: potential role in idiopathic pulmonary fibrosis. Am J Pathol, 166:1321-1332.
4. Wei WL, Zeng R, Gu CM, Qu Y, Huang LF (2016). Angelica sinensis in China-A review of botanical profile, ethnopharmacology, phytochemistry and chemical analysis. J Ethnopharmacol, 190:116-141.
5. Jin M, Zhao K, Huang Q, Xu C, Shang P (2012). Isolation, structure and bioactivities of the polysaccharides from Angelica sinensis (Oliv.) Diels: a review. Carbohydr Polym, 89:713-722.
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