Zn-dependent β-amyloid Aggregation and its Reversal by the Tetrapeptide HAEE-Reference-Cited by-同舟云学术

Zn-dependent β-amyloid Aggregation and its Reversal by the Tetrapeptide HAEE

Published:2022 Issue: Volume: Page:0
ISSN:2152-5250
Container-title:Aging and disease
language:en
Short-container-title:Aging and disease

Author:

Mitkevich Vladimir A.,Barykin Evgeny P.,Eremina Svetlana,Pani Bibhusita,Katkova-Zhukotskaya Olga,Polshakov Vladimir I.,Adzhubei Alexei A.,Kozin Sergey A.,Mironov Alexander S.,Makarov Alexander A.,Nudler Evgeny

Publisher

Aging and Disease

Subject

Cell Biology,Neurology (clinical),Geriatrics and Gerontology,Pathology and Forensic Medicine

Link

http://www.aginganddisease.org/fileup/2152-5250/PDF/1663615683831-1544945235.pdf

Reference46 articles.

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2. Barykin EP, Mitkevich VA, Kozin SA, Makarov AA (2017). Amyloid β modification: a key to the sporadic Alzheimer’s disease? Frontiers in Genetics, 8:58.

3. Kozin SA, Mitkevich VA, Makarov AA (2016). Amyloid-b containing isoaspartate 7 as potential biomarker and drug target in Alzheimer’s disease. Mendeleev Communications, 26:269-275

4. Mukherjee S, Perez KA, Lago LC, Klatt S, McLean CA, Birchall IE, et al. (2021). Quantification of N-terminal amyloid-β isoforms reveals isomers are the most abundant form of the amyloid-β peptide in sporadic Alzheimer's disease. Brain Commun, 3:fcab028.

5. Clarke S (2003). Aging as war between chemical and biochemical processes: protein methylation and the recognition of age-damaged proteins for repair. Ageing Res Rev, 2:263-285.

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