Early Metabolic Response by PET Predicts Sensitivity to Next-Line Targeted Therapy in<i>EGFR</i>-Mutated Lung Cancer with Unknown Mechanism of Acquired Resistance-Reference-Cited by-同舟云学术

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Early Metabolic Response by PET Predicts Sensitivity to Next-Line Targeted Therapy inEGFR-Mutated Lung Cancer with Unknown Mechanism of Acquired Resistance

Published:2024-04-04 Issue:6 Volume:65 Page:851-855
ISSN:0161-5505
Container-title:Journal of Nuclear Medicine
language:en
Short-container-title:J Nucl Med

Author:

Schuler Martin,Hense Jörg,Darwiche Kaid,Michels Sebastian,Hautzel Hubertus,Kobe Carsten,Lueong Smiths,Metzenmacher Martin,Herold Thomas,Zaun Gregor,Laue Katharina,Drzezga Alexander,Theegarten Dirk,Nensa Felix,Wolf Jürgen,Herrmann Ken,Wiesweg Marcel

Publisher

Society of Nuclear Medicine

Reference21 articles.

1. Genotypic and Histological Evolution of Lung Cancers Acquiring Resistance to EGFR Inhibitors

2. Circulating tumour DNA profiling reveals heterogeneity of EGFR inhibitor resistance mechanisms in lung cancer patients;Chabon;Nat Commun.,2016

3. Diverse alterations associated with resistance to KRAS(G12C) inhibition

4. Tracking the evolution of resistance to ALK tyrosine kinase inhibitors through longitudinal analysis of circulating tumor DNA;Dagogo-Jack;JCO Precis Oncol.,2018

5. Tepotinib plus gefitinib in patients with EGFR-mutant non-small-cell lung cancer with MET overexpression or MET amplification and acquired resistance to previous EGFR inhibitor (INSIGHT study): an open-label, phase 1b/2, multicentre, randomised trial;Wu;Lancet Respir Med.,2020

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