PET and MRI Reveal Early Evidence of Neurodegeneration in Spinocerebellar Ataxia Type 17-Reference-Cited by-同舟云学术

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PET and MRI Reveal Early Evidence of Neurodegeneration in Spinocerebellar Ataxia Type 17

Published:2012-05-31 Issue:7 Volume:53 Page:1074-1080
ISSN:0161-5505
Container-title:Journal of Nuclear Medicine
language:en
Short-container-title:J Nucl Med

Author:

Brockmann Kathrin,Reimold Matthias,Globas Christoph,Hauser Till Karsten,Walter Uwe,Machulla Hans-Jürgen,Rolfs Arndt,Schöls Ludger

Publisher

Society of Nuclear Medicine

Subject

Radiology, Nuclear Medicine and imaging

Reference38 articles.

1. SCA17, a novel autosomal dominant cerebellar ataxia caused by an expanded polyglutamine in TATA-binding protein

2. Spinocerebellar ataxia type 17 associated with an expansion of 42 glutamine residues in TATA-box binding protein gene

3. Clinical features and neuropathology of autosomal dominant spinocerebellar ataxia (SCA17)

4. Spinocerebellar ataxia type 17 is caused by mutations in the TATA-box binding protein;Zühlke;Cerebellum.,2007

5. Huntington's disease-like phenotype due to trinucleotide repeat expansions in the TBP and JPH3 genes

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1. ACR Appropriateness Criteria® Dizziness and Ataxia: 2023 Update;Journal of the American College of Radiology;2024-06

2. Cognitive dysfunction, social behavior disorder, cerebellar ataxia, and atypical brain FDG-PET presentation in spinocerebellar ataxia 17: a case report;Neurological Sciences;2024-03-18

3. On the Cut-Off Value of the Anteroposterior Diameter of the Midbrain Atrophy in Spinocerebellar Ataxia Type 2 Patients;Brain Sciences;2024-01-05

4. Myelinating Glia: Potential Therapeutic Targets in Polyglutamine Spinocerebellar Ataxias;Cells;2023-02-13

5. [18F]FDG PET in conditions associated with hyperkinetic movement disorders and ataxia: a systematic review;European Journal of Nuclear Medicine and Molecular Imaging;2023-01-27

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