The Senolytic Drug Navitoclax (ABT-263) Causes Trabecular Bone Loss and Impaired Osteoprogenitor Function in Aged Mice-Reference-Cited by-同舟云学术

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The Senolytic Drug Navitoclax (ABT-263) Causes Trabecular Bone Loss and Impaired Osteoprogenitor Function in Aged Mice

Published:2020-05-20 Issue: Volume:8 Page:
ISSN:2296-634X
Container-title:Frontiers in Cell and Developmental Biology
language:
Short-container-title:Front. Cell Dev. Biol.

Author:

Sharma Anuj K.,Roberts Rachel L.,Benson Reginald D.,Pierce Jessica L.,Yu Kanglun,Hamrick Mark W.,McGee-Lawrence Meghan E.

Funder

National Institute on Aging

Publisher

Frontiers Media SA

Subject

Cell Biology,Developmental Biology

Reference36 articles.

1. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders.;Baker;Nature,2011

2. The effects of dexamethasone and 1,25-dihydroxyvitamin D3 on osteogenic differentiation of human marrow stromal cells in vitro.;Beresford;Arch. Oral. Biol.,1994

3. Paradoxes of senolytics.;Blagosklonny;Aging (Albany N. Y.),2018

4. Cellular senescence: aging. cancer, and injury.;Calcinotto;Physiol. Rev.,2019

5. Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice.;Chang;Nat. Med.,2016

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