Key Role of the Membrane Trafficking of Nav1.5 Channel Protein in Antidepressant-Induced Brugada Syndrome-Reference-Cited by-同舟云学术

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Key Role of the Membrane Trafficking of Nav1.5 Channel Protein in Antidepressant-Induced Brugada Syndrome

Published:2018-09-05 Issue: Volume:9 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Chen Xi,Zhu Chao,Zhou Hao,Zhang Yu,Cai Zhongqi,Wu Honglin,Ren Xiaomeng,Gao Lei,Zhang Jiancheng,Li Yang

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Reference34 articles.

1. Ion channel macromolecular complexes in cardiomyocytes: roles in sudden cardiac death.;Abriel;Circ. Res.,2015

2. Sudden cardiac arrest associated with use of a non-cardiac drug that reduces cardiac excitability: evidence from bench, bedside and community.;Bardai;Eur. Heart J.,2013

3. Tubulin polymerization modifies cardiac sodium channel expression and gating.;Casini;Cardiovasc. Res.,2010

4. The mitochondrial calcium uniporter is involved in mitochondrial calcium cycle dysfunction: underlying mechanism of hypertension associated with mitochondrial tRNA(Ile) A4263G mutation.;Chen;Int. J. Biochem. Cell Biol.,2016

5. Brugada syndrome precipitated by a tricyclic antidepressant.;Chow;Heart,2005

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3. Amitriptyline intoxication in bullfrogs causes widening of QRS complexes in electrocardiogram;Journal of Veterinary Medical Science;2023

4. Plasma MicroRNAs as noninvasive diagnostic biomarkers in patients with Brugada syndrome;PLOS ONE;2022-05-26

5. Brugada syndrome: should we be screening patients before prescribing psychotropic medication?;Therapeutic Advances in Psychopharmacology;2022-01

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