Loss of Proteostatic Control as a Substrate for Atrial Fibrillation: A Novel Target for Upstream Therapy by Heat Shock Proteins
Author:
Publisher
Frontiers Media SA
Subject
Physiology (medical),Physiology
Cited by 20 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. The SK4 channel allosteric blocker, BA6b9, reduces atrial fibrillation substrate in rats with reduced ejection fraction;PNAS Nexus;2024-04-30
2. Small molecules as modulators of the proteostasis machinery: Implication in cardiovascular diseases;European Journal of Medicinal Chemistry;2024-01
3. Molecular mechanisms underlying sarcopenia in heart failure;The Journal of Cardiovascular Aging;2024-01-01
4. Research Priorities in the Secondary Prevention of Atrial Fibrillation: A National Heart, Lung, and Blood Institute Virtual Workshop Report;Journal of the American Heart Association;2021-08-17
5. Role of Oxidative DNA Damage and Repair in Atrial Fibrillation and Ischemic Heart Disease;International Journal of Molecular Sciences;2021-04-07
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