Reduced Neurosteroid Exposure Following Preterm Birth and Its’ Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies-Reference-Cited by-同舟云学术

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Reduced Neurosteroid Exposure Following Preterm Birth and Its’ Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies

Published:2019-05-15 Issue: Volume:10 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Shaw Julia C.,Berry Mary J.,Dyson Rebecca M.,Crombie Gabrielle K.,Hirst Jonathan J.,Palliser Hannah K.

Funder

National Health and Medical Research Council

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Reference169 articles.

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2. Human endogenous retrovirus glycoprotein-mediated induction of redox reactants causes oligodendrocyte death and demyelination.;Antony;Nat. Neurosci.,2004

3. Axon-to-Glia interaction regulates GABAA receptor expression in oligodendrocytes.;Arellano;Mol. Pharmacol.,2016

4. Human fetal hippocampal development: I. Cytoarchitecture, myeloarchitecture, and neuronal morphologic features.;Arnold;J. Comp. Neurol.,1996

5. Selective vulnerability of late oligodendrocyte progenitors to hypoxia–ischemia.;Back;J. Neurosci.,2002

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3. The influence of temperament and perinatal factors on language development: a longitudinal study;Frontiers in Psychology;2024-07-04

4. Ongoing effects of preterm birth on the dopaminergic and noradrenergic pathways in the frontal cortex and hippocampus of guinea pigs;Developmental Neurobiology;2024-03-25

5. Early IGF-1 receptor inhibition in mice mimics preterm human brain disorders and reveals a therapeutic target;Science Advances;2024-03

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