Age-Dependent Decline in Cardiac Function in Guanidinoacetate-N-Methyltransferase Knockout Mice-Reference-Cited by-同舟云学术

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Age-Dependent Decline in Cardiac Function in Guanidinoacetate-N-Methyltransferase Knockout Mice

Published:2020-01-21 Issue: Volume:10 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Aksentijević Dunja,Zervou Sevasti,Eykyn Thomas R.,McAndrew Debra J.,Wallis Julie,Schneider Jurgen E.,Neubauer Stefan,Lygate Craig A.

Funder

British Heart Foundation

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Reference64 articles.

1. Running performance and cardiovascular capacity are not impaired in creatine-depleted rats.;Adams;J. Appl. Physiol.,1995

2. The biological functions of polyamine oxidation products by amine oxidases: perspectives of clinical applications.;Agostinelli;Amino. Acids,2004

3. A Japanese adult case of guanidinoacetate methyltransferase deficiency.;Akiyama;JIMD Rep.,2014

4. High-energy phosphotransfer in the failing mouse heart: role of adenylate kinase and glycolytic enzymes.;Aksentijevic;Eur. J. Heart Fail.,2010

5. Cardiac dysfunction and peri-weaning mortality in malonyl-coenzyme A decarboxylase (MCD) knockout mice as a consequence of restricting substrate plasticity.;Aksentijevic;J. Mol. Cell Cardiol.

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