Whole Genome Expression Analysis in a Mouse Model of Tauopathy Identifies MECP2 as a Possible Regulator of Tau Pathology-Reference-Cited by-同舟云学术

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Whole Genome Expression Analysis in a Mouse Model of Tauopathy Identifies MECP2 as a Possible Regulator of Tau Pathology

Published:2017-03-17 Issue: Volume:10 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Maphis Nicole M.,Jiang Shanya,Binder Jessica,Wright Carrie,Gopalan Banu,Lamb Bruce T.,Bhaskar Kiran

Funder

National Institute of Neurological Disorders and Stroke

BrightFocus Foundation

Alzheimer’s Association

National Institute on Aging

U.S. Department of Defense

National Science Foundation

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference48 articles.

1. Microarray analysis of CA1 pyramidal neurons in a mouse model of tauopathy reveals progressive synaptic dysfunction.;Alldred;Neurobiol. Dis.,2012

2. Gene expression, methylation and neuropathology correlations at progressive supranuclear palsy risk loci.;Allen;Acta Neuropathol.,2016

3. Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2.;Amir;Nat. Genet.,1999

4. Cell-cycle reentry and cell death in transgenic mice expressing nonmutant human tau isoforms.;Andorfer;J. Neurosci.,2005

5. Hyperphosphorylation and aggregation of tau in mice expressing normal human tau isoforms.;Andorfer;J. Neurochem.,2003

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