Cosyntropin Attenuates Neuroinflammation in a Mouse Model of Traumatic Brain Injury-Reference-Cited by-同舟云学术

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Cosyntropin Attenuates Neuroinflammation in a Mouse Model of Traumatic Brain Injury

Published:2020-06-26 Issue: Volume:13 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Siebold Lorraine,Krueger Amy C.,Abdala Jonathan A.,Figueroa Johnny D.,Bartnik-Olson Brenda,Holshouser Barbara,Wilson Christopher G.,Ashwal Stephen

Funder

Foundation for the National Institutes of Health

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference113 articles.

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2. Absence of TLR4 reduces neurovascular unit and secondary inflammatory process after traumatic brain injury in mice;Ahmad;PLoS One,2013

3. C-terminal fragments of ACTH stimulate feeding in fasted rats;Al-Barazanji;Horm. Metab. Res.,2001

4. The novel object recognition memory: neurobiology, test procedure, and its modifications;Antunes;Cogn. Process.,2012

5. Post-traumatic epilepsy in children requiring inpatient rehabilitation following head injury;Appleton;J. Neurol. Neurosurg. Psychiatry,2002

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1. Autophagy machinery plays an essential role in traumatic brain injury-induced apoptosis and its related behavioral abnormalities in mice: focus on Boswellia Sacra gum resin;Frontiers in Physiology;2024-01-05

2. Bumetanide induces post-traumatic microglia–interneuron contact to promote neurogenesis and recovery;Brain;2023-04-21

3. Traumatic brain injury and the development of parkinsonism: Understanding pathophysiology, animal models, and therapeutic targets;Biomedicine & Pharmacotherapy;2022-05

4. Post-traumatic Neuroinflammation: Relevance to Pediatrics;Pediatric Neurology;2021-09

5. Atrazine Inhalation Causes Neuroinflammation, Apoptosis and Accelerating Brain Aging;International Journal of Molecular Sciences;2021-07-26

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