Chronic Sodium Selenate Treatment Restores Deficits in Cognition and Synaptic Plasticity in a Murine Model of Tauopathy-Reference-Cited by-同舟云学术

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Chronic Sodium Selenate Treatment Restores Deficits in Cognition and Synaptic Plasticity in a Murine Model of Tauopathy

Published:2020-10-08 Issue: Volume:13 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Ahmed Tariq,Van der Jeugd Ann,Caillierez Raphaëlle,Buée Luc,Blum David,D’Hooge Rudi,Balschun Detlef

Funder

Fonds Wetenschappelijk Onderzoek

Association France Alzheimer

Development of Innovative Strategies for a Transdisciplinary approach to ALZheimer’s disease

Agence Nationale de la Recherche

Fondation pour la Recherche Médicale

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference100 articles.

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2. An N-methyl-D-aspartate-receptor dependent, late-phase long-term depression in middle-aged mice identifies no GluN2-subunit bias;Ahmed;Neuroscience,2011

3. Specific tau phosphorylation sites correlate with severity of neuronal cytopathology in Alzheimer’s disease;Augustinack;Acta Neuropathol.,2002

4. Bizarre behaviors and risk assessment in 3xTg-AD mice at early stages of the disease;Baeta-Corral;Behav. Brain Res.,2014

5. Does cAMP response element-binding protein have a pivotal role in hippocampal synaptic plasticity and hippocampus-dependent memory?;Balschun;J. Neurosci,2003

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