Myosin IIA-related Actomyosin Contractility Mediates Oxidative Stress-induced Neuronal Apoptosis-Reference-Cited by-同舟云学术

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Myosin IIA-related Actomyosin Contractility Mediates Oxidative Stress-induced Neuronal Apoptosis

Published:2017-03-14 Issue: Volume:10 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Wang Yan,Xu Yingqiong,Liu Qian,Zhang Yuanyuan,Gao Zhen,Yin Mingzhu,Jiang Nan,Cao Guosheng,Yu Boyang,Cao Zhengyu,Kou Junping

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference72 articles.

1. Cytochalasin D disruption of actin filaments in 3T3 cells produces an anti-apoptotic response by activating gelatinase A extracellularly and initiating intracellular survival signals;Ailenberg;Biochim. Biophys. Acta,2003

2. The structural basis of blebbistatin inhibition and specificity for myosin II;Allingham;Nat. Struct. Mol. Biol.,2005

3. Replacement of nonmuscle myosin II-B with II-A rescues brain but not cardiac defects in mice;Bao;J. Biol. Chem.,2007

4. Myosin II isoform co-assembly and differential regulation in mammalian systems;Beach;Exp. Cell Res.,2015

5. Caspases 3 and 9 are translocated to the cytoskeleton and activated by thrombin in human platelets. Evidence for the involvement of PKC and the actin filament polymerization;Ben Amor;Cell. Signal.,2006

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