Accumulation of Cav3.2 T-type Calcium Channels in the Uninjured Sural Nerve Contributes to Neuropathic Pain in Rats with Spared Nerve Injury-Reference-Cited by-同舟云学术

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Accumulation of Cav3.2 T-type Calcium Channels in the Uninjured Sural Nerve Contributes to Neuropathic Pain in Rats with Spared Nerve Injury

Published:2018-02-08 Issue: Volume:11 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Chen Wen,Chi Ye-Nan,Kang Xue-Jing,Liu Qing-Ying,Zhang Hao-Lin,Li Zhi-Hua,Zhao Zi-Fang,Yang Yin,Su Li,Cai Jie,Liao Fei-Fei,Yi Ming,Wan You,Liu Feng-Yu

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference68 articles.

1. Uninjured C-fiber nociceptors develop spontaneous activity and alpha- adrenergic sensitivity following L6 spinal nerve ligation in monkey;Ali;J. Neurophysiol.,1999

2. Investigation of pain mechanisms by calcium imaging approaches;Anderson;Neurosci. Bull.,2017

3. Genetic tracing of Cav3.2 T-type calcium channel expression in the peripheral nervous system;Bernal Sierra;Front. Mol. Neurosci.,2017

4. Silencing of the Cav3.2 T-type calcium channel gene in sensory neurons demonstrates its major role in nociception;Bourinet;EMBO J.,2005

5. T-type calcium channels in neuropathic pain;Bourinet;Pain,2016

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