Increasing N-acetylaspartate in the Brain during Postnatal Myelination Does Not Cause the CNS Pathologies of Canavan Disease-Reference-Cited by-同舟云学术

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Increasing N-acetylaspartate in the Brain during Postnatal Myelination Does Not Cause the CNS Pathologies of Canavan Disease

Published:2017-06-02 Issue: Volume:10 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Appu Abhilash P.,Moffett John R.,Arun Peethambaran,Moran Sean,Nambiar Vikram,Krishnan Jishnu K. S.,Puthillathu Narayanan,Namboodiri Aryan M. A.

Funder

National Institutes of Health

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference83 articles.

1. Spongy degeneration of the central nervous system (van Bogaert and Bertrand type; Canavan’s disease). A review;Adachi;Hum. Pathol.,1973

2. A single intravenous rAAV injection as late as P20 achieves efficacious and sustained CNS gene therapy in canavan mice;Ahmed;Mol. Ther.,2013

3. rAAV gene therapy in a Canavan’s disease mouse model reveals immune impairments and an extended pathology beyond the central nervous system;Ahmed;Mol. Ther.,2016

4. Epileptic seizures induced by N-acetyl-L-aspartate in rats: in vivo and in vitro studies;Akimitsu;Brain Res.,2000

5. Quantification of N-acetylaspartic acid in urine by LC-MS/MS for the diagnosis of Canavan disease;Al-Dirbashi;J. Inherit. Metab. Dis.,2007

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1. Cellular and molecular mechanisms of aspartoacylase and its role in Canavan disease;Cell & Bioscience;2024-04-06

2. N-acetyl-aspartate metabolism at the interface of cancer, immunity, and neurodegeneration;Current Opinion in Biotechnology;2024-02

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4. Pathological Bergmann glia alterations and disrupted calcium dynamics in ataxic Canavan disease mice;Glia;2023-08-23

5. Renewal of oligodendrocyte lineage reverses dysmyelination and CNS neurodegeneration through corrected N-acetylaspartate metabolism;Progress in Neurobiology;2023-07

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