Neuronal Gene Targets of NF-κB and Their Dysregulation in Alzheimer's Disease-Reference-Cited by-同舟云学术

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Neuronal Gene Targets of NF-κB and Their Dysregulation in Alzheimer's Disease

Published:2016-11-09 Issue: Volume:9 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Snow Wanda M.,Albensi Benedict C.

Funder

Natural Sciences and Engineering Research Council of Canada

St. Boniface General Hospital Research Foundation

Research Manitoba

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference223 articles.

1. Brain in situ hybridization maps as a source for reverse-engineering transcriptional regulatory networks: Alzheimer's disease insights;Acquaah-Mensah;Gene,2016

2. Astrocytes protect neurons from Aβ1-42 peptide-induced neurotoxicity increasing TFAM and PGC-1 and decreasing PPAR-gamma and SIRT-1;Aguirre-Rueda;Int. J. Med. Sci.,2015

3. c-Rel, an NF-κB family transcription factor, is required for hippocampal long-term synaptic plasticity and memory formation;Ahn;Learn. Mem.,2008

4. Inflammatory mechanisms in Alzheimer's disease: implications for therapy;Aisen;Am. J. Psychiatry,1994

5. Evidence for the involvement of TNF and NF-kappaB in hippocampal synaptic plasticity;Albensi;Synapse,2000

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