Laminin and Integrin in LAMA2-Related Congenital Muscular Dystrophy: From Disease to Therapeutics-Reference-Cited by-同舟云学术

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Laminin and Integrin in LAMA2-Related Congenital Muscular Dystrophy: From Disease to Therapeutics

Published:2020-02-11 Issue: Volume:13 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Barraza-Flores Pamela,Bates Christina R.,Oliveira-Santos Ariany,Burkin Dean J.

Funder

National Institutes of Health

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference80 articles.

1. Integrin dysregulation as a possible driver of matrix remodeling in laminin-deficient congenital muscular dystrophy (MDC1A);Accorsi;J. Neuromuscul. Dis.,2015

2. Laminin-111 protein therapy enhances muscle regeneration and repair in the GRMD dog model of duchenne muscular dystrophy;Barraza-Flores;Hum. Mol. Genet.,2019

3. Muscle B1D integrin reinforces the cytoskeleton-matrix link: modulation of integrin adhesive function by alternative splicing;Belkin;J. Cell Biol.,1997

4. Integrin signaling: linking mechanical stimulation to skeletal muscle hypertrophy;Boppart;Am. J. Physiol. Cell Physiol.,2019

5. The α7β1 integrin in muscle development and disease;Burkin;Cell Tissue Res.,1999

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