A Decade of Boon or Burden: What Has the CHIP Ever Done for Cellular Protein Quality Control Mechanism Implicated in Neurodegeneration and Aging?-Reference-Cited by-同舟云学术

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A Decade of Boon or Burden: What Has the CHIP Ever Done for Cellular Protein Quality Control Mechanism Implicated in Neurodegeneration and Aging?

Published:2016-10-04 Issue: Volume:9 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Joshi Vibhuti,Amanullah Ayeman,Upadhyay Arun,Mishra Ribhav,Kumar Amit,Mishra Amit

Funder

Science and Engineering Research Board

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference192 articles.

1. CHIP overexpression reduces mutant androgen receptor protein and ameliorates phenotypes of the spinal and bulbar muscular atrophy transgenic mouse model;Adachi;J. Neurosci.,2007

2. Aβ, tau and ApoE4 in Alzheimer’s disease: the axonal connection;Adalbert;Trends Mol. Med.,2007

3. The chaperone-assisted E3 ligase C terminus of Hsc70-interacting protein (CHIP) targets PTEN for proteasomal degradation;Ahmed;J. Biol. Chem.,2012

4. CHIP protects from the neurotoxicity of expanded and wild-type ataxin-1 and promotes their ubiquitination and degradation;Al-Ramahi;J. Biol. Chem.,2006

5. The U box is a modified RING finger–a common domain in ubiquitination;Aravind;Curr. Biol.,2000

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3. Graphic contrastive learning analyses of discontinuous molecular dynamics simulations: Study of protein folding upon adsorption;Applied Physics Letters;2023-06-19

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5. KDM1A-mediated upregulation of METTL3 ameliorates Alzheimer's disease via enhancing autophagic clearance of p-Tau through m6A-dependent regulation of STUB1;Free Radical Biology and Medicine;2023-02

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