An APC:WNT Counter-Current-Like Mechanism Regulates Cell Division Along the Human Colonic Crypt Axis: A Mechanism That Explains How APC Mutations Induce Proliferative Abnormalities That Drive Colon Cancer Development-Reference-Cited by-同舟云学术

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An APC:WNT Counter-Current-Like Mechanism Regulates Cell Division Along the Human Colonic Crypt Axis: A Mechanism That Explains How APC Mutations Induce Proliferative Abnormalities That Drive Colon Cancer Development

Published:2013 Issue: Volume:3 Page:
ISSN:2234-943X
Container-title:Frontiers in Oncology
language:
Short-container-title:Front. Oncol.

Author:

Boman Bruce M.,Fields Jeremy Z.

Publisher

Frontiers Media SA

Subject

Cancer Research,Oncology

Cited by 51 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. CYP26A1 Links WNT and Retinoic Acid Signaling: A Target to Differentiate ALDH+ Stem Cells in APC-Mutant CRC;Cancers;2024-01-07

2. Regulation and functions of cell division in the intestinal tissue;Seminars in Cell & Developmental Biology;2023-12

3. Beyond the Genetic Code:A Tissue Code?;2023-03-06

4. A Case of Sporadic Multiple Colonic Polyps in a Young Woman;Current Oncology;2023-01-17

5. Mathematical model for simulation of morphological changes associated to crypt fission in the colon;Discrete and Continuous Dynamical Systems - S;2022

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