Author:
Hegde Anisha,Denburg Michelle R.,Glenn Dorey A.
Abstract
Acute kidney injury (AKI) has been associated with deleterious impacts on a variety of body systems. While AKI is often accompanied by dysregulation of mineral metabolism—including alterations in calcium, phosphate, vitamin D, parathyroid hormone, fibroblast growth factor 23, and klotho—its direct effects on the skeletal system of children and adolescents remain largely unexplored. In this review, the pathophysiology of dysregulated mineral metabolism in AKI and its potential effects on skeletal health are discussed, including data associating AKI with fracture risk.
Subject
Pediatrics, Perinatology, and Child Health
Reference94 articles.
1. Chapter 365: Acute kidney injury
GoldsteinSL
AdrogueHE
McInernyT
AdamH
CampbellD
DeWittT
FoyJ
KamatD
American Academy of Pediatrics Textbook of Pediatric Care2017
2. Epidemiology of acute kidney injury in children worldwide, including developing countries;Lameire;Pediatr Nephrol.,2017
3. KDIGO clinical practice guidelines for acute kidney injury;Khwaja;Nephron Clin Pract.,2012
4. World incidence of AKI: A meta-analysis;Susantitaphong;Clin J Am Soc Nephrol.,2013
5. The aftermath of acute kidney injury: a narrative review of long-term mortality and renal function;Fortrie;Crit Care.,2019
Cited by
6 articles.
订阅此论文施引文献
订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献