Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: a model for fetal alcohol spectrum disorders-Reference-Cited by-同舟云学术

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Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: a model for fetal alcohol spectrum disorders

Published:2014-06-02 Issue: Volume:5 Page:
ISSN:1664-8021
Container-title:Frontiers in Genetics
language:
Short-container-title:Front. Genet.

Author:

Kleiber Morgan L.,Diehl Eric J.,Laufer Benjamin I.,Mantha Katarzyna,Chokroborty-Hoque Aniruddho,Alberry Bonnie,Singh Shiva M.

Publisher

Frontiers Media SA

Subject

Genetics(clinical),Genetics,Molecular Medicine

Reference100 articles.

1. A mouse model of prenatal ethanol exposure using a voluntary drinking paradigm;Allan;Alcohol. Clin. Exp. Res,2003

2. Betaine effects on hepatic methionine metabolism elicited by short-term ethanol feeding;Barak;Alcohol,1996

3. High-resolution profiling of histone methylations in the human genome;Barski;Cell,2007

4. Gestational choline supplementation normalized fetal alcohol-induced alterations in histone modifications, DNA methylation, and proopiomelanocortin (POMC) gene expression in beta-endorphin-producing POMC neurons of the hypothalamus;Bekdash;Alcohol. Clin. Exp. Res,2013

5. Using drinking in the dark to model prenatal binge-like exposure to ethanol in C57BL/6J mice;Boehm;Dev. Psychobiol,2008

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