Mitochondria: A Common Target for Genetic Mutations and Environmental Toxicants in Parkinson’s Disease-Reference-Cited by-同舟云学术

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Mitochondria: A Common Target for Genetic Mutations and Environmental Toxicants in Parkinson’s Disease

Published:2017-11-17 Issue: Volume:8 Page:
ISSN:1664-8021
Container-title:Frontiers in Genetics
language:
Short-container-title:Front. Genet.

Author:

Helley Martin P.,Pinnell Jennifer,Sportelli Carolina,Tieu Kim

Funder

National Institute of Environmental Health Sciences

Publisher

Frontiers Media SA

Subject

Genetics(clinical),Genetics,Molecular Medicine

Reference326 articles.

1. Increased mitochondrial calcium sensitivity and abnormal expression of innate immunity genes precede dopaminergic defects in Pink1-deficient mice.;Akundi;PLOS ONE,2011

2. Deregulation of mitochondria-shaping proteins Opa-1 and Drp-1 in manganese-induced apoptosis.;Alaimo;PLOS ONE,2014

3. Toxicokinetics of deltamethrin and its 4’-HO-metabolite in the rat.;Anadón;Toxicol. Appl. Pharmacol.,1996

4. Alpha-synuclein p.H50q, a novel pathogenic mutation for Parkinson’s disease.;Appel-Cresswell;Mov. Disord.,2013

5. MNRR1 (formerly CHCHD2) is a bi-organellar regulator of mitochondrial metabolism.;Aras;Mitochondrion,2015

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