Alpha5 Nicotinic Acetylcholine Receptor Contributes to Nicotine-Induced Lung Cancer Development and Progression-Reference-Cited by-同舟云学术

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Alpha5 Nicotinic Acetylcholine Receptor Contributes to Nicotine-Induced Lung Cancer Development and Progression

Published:2017-08-23 Issue: Volume:8 Page:
ISSN:1663-9812
Container-title:Frontiers in Pharmacology
language:
Short-container-title:Front. Pharmacol.

Author:

Sun Hai-Ji,Jia Yan-Fei,Ma Xiao-Li

Funder

National Natural Science Foundation of China

Natural Science Foundation of Shandong Province

Publisher

Frontiers Media SA

Subject

Pharmacology (medical),Pharmacology

Reference49 articles.

1. Effects of chronic nicotine on the autocrine regulation of pancreatic cancer cells and pancreatic duct epithelial cells by stimulatory and inhibitory neurotransmitters.;Al-Wadei;Carcinogenesis,2012

2. Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog signalling.;Al-Wadei;Eur. J. Cancer,2016

3. Genome-wide association scan of tag SNPs identifies a susceptibility locus for lung cancer at 15q25.1.;Amos;Nat. Genet.,2008

4. Receptor-mediated tobacco toxicity: cooperation of the Ras/Raf-1/MEK1/ERK and JAK-2/STAT-3 pathways downstream of alpha7 nicotinic receptor in oral keratinocytes.;Arredondo;FASEB J.,2006

5. In silico analyses reveal common cellular pathways affected by loss of heterozygosity (LOH) events in the lymphomagenesis of Non-Hodgkin’s lymphoma (NHL).;Aya-Bonilla;BMC Genomics,2014

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