GYY4137 Promotes Mice Feeding Behavior via Arcuate Nucleus Sulfur-Sulfhydrylation and AMPK Activation-Reference-Cited by-同舟云学术

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GYY4137 Promotes Mice Feeding Behavior via Arcuate Nucleus Sulfur-Sulfhydrylation and AMPK Activation

Published:2018-08-21 Issue: Volume:9 Page:
ISSN:1663-9812
Container-title:Frontiers in Pharmacology
language:
Short-container-title:Front. Pharmacol.

Author:

Zhou Jun,Lv Xiao-Hui,Fan Jun-Juan,Dang Li-Yun,Dong Kun,Gao Bo,Song Ao-Qi,Wu Wen-Ning

Funder

National Natural Science Foundation of China

Natural Science Foundation of Shaanxi Province

Publisher

Frontiers Media SA

Subject

Pharmacology (medical),Pharmacology

Reference48 articles.

1. Hypothalamic CaMKK2 contributes to the regulation of energy balance.;Anderson;Cell Metab.,2008

2. Differential expression of feeding-related hypothalamic neuropeptides in the first generation of quails divergently selected for low or high feed efficiency.;Blankenship;Neuropeptides,2016

3. Evidence for hypothalamic ketone body sensing: impact on food intake and peripheral metabolic responses in mice.;Carneiro;Am. J. Physiol. Endocrinol. Metab.,2016

4. Hydrogen sulphide treatment increases insulin sensitivity and improves oxidant pathway in PA-induced IR C2C12 cells metabolism through the CaMKKbeta-AMPK.;Chen;Sci. Rep.,2017

5. Lowering glucose level elevates [Ca2+]i in hypothalamic arcuate nucleus NPY neurons through P/Q-type Ca2+ channel activation and GSK3β inhibition.;Chen;Acta Pharmacol. Sin.,2012

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