Nrf2 as a Potential Mediator of Cardiovascular Risk in Metabolic Diseases-Reference-Cited by-同舟云学术

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Nrf2 as a Potential Mediator of Cardiovascular Risk in Metabolic Diseases

Published:2019-04-12 Issue: Volume:10 Page:
ISSN:1663-9812
Container-title:Frontiers in Pharmacology
language:
Short-container-title:Front. Pharmacol.

Author:

da Costa Rafael M.,Rodrigues Daniel,Pereira Camila A.,Silva Josiane F.,Alves Juliano V.,Lobato Núbia S.,Tostes Rita C.

Funder

Fundação de Amparo à Pesquisa do Estado de São Paulo

Publisher

Frontiers Media SA

Subject

Pharmacology (medical),Pharmacology

Reference122 articles.

1. Discovery of direct inhibitors of Keap1–Nrf2 protein–protein interaction as potential therapeutic and preventive agents.;Abed;Acta Pharm. Sin. B,2015

2. Hydrogen sulfide attenuates calcification of vascular smooth muscle cells via KEAP1/NRF2/NQO1 activation.;Aghagolzadeh;Atherosclerosis,2017

3. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and rho kinase-dependent mechanisms.;Alves-Lopes;Hypertension,2016

4. Nrf2/Keap1 system regulates vascular smooth muscle cell apoptosis for vascular homeostasis: role in neointimal formation after vascular injury.;Ashino;Sci. Rep.,2016

5. Redox-sensitive transcription factor Nrf2 regulates vascular smooth muscle cell migration and neointimal hyperplasia significance.;Ashino;Arterioscler. Thromb. Vasc. Biol.,2013

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