Qi-Li-Qiang-Xin Alleviates Isoproterenol-Induced Myocardial Injury by Inhibiting Excessive Autophagy via Activating AKT/mTOR Pathway-Reference-Cited by-同舟云学术

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Qi-Li-Qiang-Xin Alleviates Isoproterenol-Induced Myocardial Injury by Inhibiting Excessive Autophagy via Activating AKT/mTOR Pathway

Published:2019-11-12 Issue: Volume:10 Page:
ISSN:1663-9812
Container-title:Frontiers in Pharmacology
language:
Short-container-title:Front. Pharmacol.

Author:

Fan Cailian,Tang Xiyang,Ye Mengnan,Zhu Guonian,Dai Yi,Yao Zhihong,Yao Xinsheng

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Pharmacology (medical),Pharmacology

Reference46 articles.

1. Isoproterenol-induced myocardial fibrosis in relation to myocyte necrosis;Benjamin;Circ. Res.,1989

2. Autophagy in the pathogenesis of disease;Beth;Cell.,2008

3. β-Adrenergic function in heart muscle disease and heart failure;Bristow;J. Mol. Cell Cardiol.,1985

4. Salidroside suppressing LPS-induced myocardial injury by inhibiting ROS-mediated PI3K/Akt/mTOR pathway in vitro and in vivo;Chen;J. Cell Mol. Med.,2017

5. Requirement of nuclear factor-kappaB in angiotensin II- and isoproterenol-induced cardiac hypertrophy in vivo;Christian;Circulation,2005

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