Deficits in Motor Performance, Neurotransmitters and Synaptic Plasticity in Elderly and Experimental Parkinsonian Mice Lacking GPR37-Reference-Cited by-同舟云学术

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Deficits in Motor Performance, Neurotransmitters and Synaptic Plasticity in Elderly and Experimental Parkinsonian Mice Lacking GPR37

Published:2020-03-27 Issue: Volume:12 Page:
ISSN:1663-4365
Container-title:Frontiers in Aging Neuroscience
language:
Short-container-title:Front. Aging Neurosci.

Author:

Zhang Xiaoqun,Mantas Ioannis,Fridjonsdottir Elva,Andrén Per E.,Chergui Karima,Svenningsson Per

Funder

European Research Council

Vetenskapsrådet

Hjärnfonden

Parkinsonfonden

Science for Life Laboratory

Publisher

Frontiers Media SA

Subject

Cognitive Neuroscience,Ageing

Reference58 articles.

1. GPR37 regulates macrophage phagocytosis and resolution of inflammatory pain.;Bang;J. Clin. Invest.,2018

2. Dopamine-mediated regulation of corticostriatal synaptic plasticity.;Calabresi;Trends Neurosci.,2007

3. Neuroprotection by caffeine and A(2A) adenosine receptor inactivation in a model of Parkinson’s disease.;Chen;J. Neurosci.,2001

4. Adenosine A2A receptor blockade prevents memory dysfunction caused by β-amyloid peptides but not by scopolamine or MK-801.;Cunha;Exp. Neurol.,2008

5. GPR37 surface expression enhancement via N-terminal truncation or protein-protein interactions.;Dunham;Biochemistry,2009

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