Pioglitazone Reduces β Amyloid Levels via Inhibition of PPARγ Phosphorylation in a Neuronal Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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Pioglitazone Reduces β Amyloid Levels via Inhibition of PPARγ Phosphorylation in a Neuronal Model of Alzheimer’s Disease

Published:2019-07-17 Issue: Volume:11 Page:
ISSN:1663-4365
Container-title:Frontiers in Aging Neuroscience
language:
Short-container-title:Front. Aging Neurosci.

Author:

Quan Qiankun,Qian Yihua,Li Xi,Li Ming

Funder

China Postdoctoral Science Foundation

Natural Science Foundation of Shaanxi Province

Publisher

Frontiers Media SA

Subject

Cognitive Neuroscience,Ageing

Reference66 articles.

1. Amyloid-β and tau: the trigger and bullet in Alzheimer disease pathogenesis;Bloom;JAMA. Neurol.,2014

2. Amyloid β-peptide (1–42)-induced oxidative stress in Alzheimer disease: importance in disease pathogenesis and progression;Butterfield;Antioxid. Redox Signal.,2013

3. Influence of drug transporters and stereoselectivity on the brain penetration of pioglitazone as a potential medicine against Alzheimer’s disease;Chang;Sci. Rep.,2015

4. Anti-diabetes drug pioglitazone ameliorates synaptic defects in AD transgenic mice by inhibiting cyclin-dependent kinase5 activity;Chen;PLoS One,2015

5. Rosiglitazone activation of PPARγ-dependent pathways is neuroprotective in human neural stem cells against amyloid-β-induced mitochondrial dysfunction and oxidative stress;Chiang;Neurobiol. Aging,2016

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