Extracellular HIV-1 Tat Mediates Increased Glutamate in the CNS Leading to Onset of Senescence and Progression of HAND-Reference-Cited by-同舟云学术

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Extracellular HIV-1 Tat Mediates Increased Glutamate in the CNS Leading to Onset of Senescence and Progression of HAND

Published:2020-06-09 Issue: Volume:12 Page:
ISSN:1663-4365
Container-title:Frontiers in Aging Neuroscience
language:
Short-container-title:Front. Aging Neurosci.

Author:

Marino Jamie,Wigdahl Brian,Nonnemacher Michael R.

Funder

National Institute of Neurological Disorders and Stroke

National Institute of Mental Health

Publisher

Frontiers Media SA

Subject

Cognitive Neuroscience,Ageing

Reference69 articles.

1. HIV-1 Tat: role in bystander toxicity.;Ajasin;Front. Cell. Infect. Microbiol.,2020

2. Factors controlling permeability of the blood-brain barrier.;Almutairi;Cell. Mol. Life Sci.,2016

3. The NMDA and AMPA/KA receptors are involved in glutamate-induced alterations of occludin expression and phosphorylation in brain endothelial cells.;Andras;J. Cereb. Blood Flow Metab.,2007

4. Signaling mechanisms of HIV-1 Tat-induced alterations of claudin-5 expression in brain endothelial cells.;Andras;J. Cereb. Blood Flow Metab.,2005

5. Effect of human immunodeficiency virus on blood-brain barrier integrity and function: an update.;Atluri;Front. Cell. Neurosci.,2015

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