Mild Traumatic Brain Injury Produces Neuron Loss That Can Be Rescued by Modulating Microglial Activation Using a CB2 Receptor Inverse Agonist-Reference-Cited by-同舟云学术

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Mild Traumatic Brain Injury Produces Neuron Loss That Can Be Rescued by Modulating Microglial Activation Using a CB2 Receptor Inverse Agonist

Published:2016-10-06 Issue: Volume:10 Page:
ISSN:1662-453X
Container-title:Frontiers in Neuroscience
language:
Short-container-title:Front. Neurosci.

Author:

Bu Wei,Ren Huiling,Deng Yunping,Del Mar Nobel,Guley Natalie M.,Moore Bob M.,Honig Marcia G.,Reiner Anton

Funder

National Institutes of Health

University of Tennessee Health Science Center

Publisher

Frontiers Media SA

Subject

General Neuroscience

Reference116 articles.

1. Reduced GABAergic inhibition in the basolateral amygdala and the development of anxiety-like behaviors after mild traumatic brain injury;Almeida-Suhett;PLoS ONE,2014

2. Cannabinoid receptor type-2 stimulation, blockade, and deletion alter the vascular inflammatory responses to traumatic brain injury;Amenta;J. Neuroinflammation,2014

3. A cannabinoid type-2 receptor agonist attenuates blood-brain barrier damage and neurodegeneration in a murine model of traumatic brain injury;Amenta;J. Neurosci.,2012

4. The cannabinoid CB2 receptor as a target for inflammation-dependent neurodegeneration;Ashton;Curr. Neuropharmacol.,2007

5. CB2 therapeutic target-in-waiting;Atwood;Prog. Neuropsychopharmacol. Biol. Psychiat.,2012

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4. Flipping the GPCR Switch: Structure-Based Development of Selective Cannabinoid Receptor 2 Inverse Agonists;ACS Central Science;2024-03-11

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