Neuroprotection Targeting Protein Misfolding on Chronic Cerebral Hypoperfusion in the Context of Metabolic Syndrome-Reference-Cited by-同舟云学术

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Neuroprotection Targeting Protein Misfolding on Chronic Cerebral Hypoperfusion in the Context of Metabolic Syndrome

Published:2018-05-31 Issue: Volume:12 Page:
ISSN:1662-453X
Container-title:Frontiers in Neuroscience
language:
Short-container-title:Front. Neurosci.

Author:

Herrera María I.,Udovin Lucas D.,Toro-Urrego Nicolás,Kusnier Carlos F.,Luaces Juan P.,Otero-Losada Matilde,Capani Francisco

Funder

Consejo Nacional de Investigaciones Científicas y Técnicas

Universidad de Buenos Aires

Fondo para la Investigación Científica y Tecnológica

Publisher

Frontiers Media SA

Subject

General Neuroscience

Reference93 articles.

1. Standards of medical care for patients with diabetes mellitus;Diabetes Care,2002

2. Candidate genes that affect aging through protein homeostasis;Argon;Adv. Exp. Med. Biol,2015

3. Protein misfolding and aggregation in Alzheimer's disease and type 2 diabetes mellitus;Ashraf;CNS Neurol. Disord. Drug Targets,2014

4. Effects of hypoperfusion in Alzheimer's disease;Austin;J. Alzheimers Dis.,2011

5. In vivo aspects of protein folding and quality control;Balchin;Science,2016

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1. Neuroprotection from protein misfolding in cerebral hypoperfusion concurrent with metabolic syndrome. A translational perspective;Frontiers in Neuroscience;2023-08-15

2. Editorial: Neurological comorbidity in metabolic syndrome;Frontiers in Neuroscience;2023-08-15

3. The role of oxidative stress in the development of Alzheimer's disease;Neurology, Neuropsychiatry, Psychosomatics;2022-08-23

4. Stress Response Is the Main Trigger of Sporadic Amyloidoses;International Journal of Molecular Sciences;2021-04-15

5. Triptolide protects against white matter injury induced by chronic cerebral hypoperfusion in mice;Acta Pharmacologica Sinica;2021-04-06

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