Loss of Fractalkine Signaling Exacerbates Axon Transport Dysfunction in a Chronic Model of Glaucoma-Reference-Cited by-同舟云学术

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Loss of Fractalkine Signaling Exacerbates Axon Transport Dysfunction in a Chronic Model of Glaucoma

Published:2016-11-24 Issue: Volume:10 Page:
ISSN:1662-453X
Container-title:Frontiers in Neuroscience
language:
Short-container-title:Front. Neurosci.

Author:

Breen Kevin T.,Anderson Sarah R.,Steele Michael R.,Calkins David J.,Bosco Alejandra,Vetter Monica L.

Funder

National Institutes of Health

Glaucoma Research Foundation

Publisher

Frontiers Media SA

Subject

General Neuroscience

Reference88 articles.

1. GpnmbR150X allele must be present in bone marrow derived cells to mediate DBA/2J glaucoma;Anderson;BMC Genet.,2008

2. Mutations in genes encoding melanosomal proteins cause pigmentary glaucoma in DBA/2J mice;Anderson;Nat. Genet.,2002

3. Progressive loss of retinal ganglion cell function precedes structural loss by several years in glaucoma suspects;Banitt;Invest. Ophthalmol. Vis. Sci.,2013

4. Glial coverage in the optic nerve expands in proportion to optic axon loss in chronic mouse glaucoma;Bosco;Exp. Eye Res,2016

5. Early reduction of microglia activation by irradiation in a model of chronic glaucoma;Bosco;PLoS ONE,2012

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