Cooperation between HMGA1, PDX-1, and MafA is Essential for Glucose-Induced Insulin Transcription in Pancreatic Beta Cells-Reference-Cited by-同舟云学术

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Cooperation between HMGA1, PDX-1, and MafA is Essential for Glucose-Induced Insulin Transcription in Pancreatic Beta Cells

Published:2015-01-13 Issue: Volume:5 Page:
ISSN:1664-2392
Container-title:Frontiers in Endocrinology
language:
Short-container-title:Front. Endocrinol.

Author:

Arcidiacono Biagio,Iiritano Stefania,Chiefari Eusebio,Brunetti Francesco S.,Gu Guoqiang,Foti Daniela Patrizia,Brunetti Antonio

Publisher

Frontiers Media SA

Subject

Endocrinology, Diabetes and Metabolism

Reference60 articles.

1. Recent advances in the molecular genetics of type 2 diabetes mellitus;Brunetti;World J Diabetes,2014

2. Perspectives on the contribution of genetics to the pathogenesis of type 2 diabetes mellitus;Brunetti;Recenti Prog Med,2011

3. Regulation of insulin synthesis and secretion and pancreatic beta-cell dysfunction in diabetes;Fu;Curr Diabetes Rev,2013

4. Glucose activates a protein phosphatase-1-mediated signaling pathway to enhance overall translation in pancreatic beta-cells;Vander Mierde;Endocrinology,2007

5. Glucose-induced transcription of the insulin gene is mediated by factors required for beta-cell-type-specific expression;Sharma;Mol Cell Biol,1994

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