Association of Clinical Phenotypes in Haploinsufficiency A20 (HA20) With Disrupted Domains of A20-Reference-Cited by-同舟云学术

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Association of Clinical Phenotypes in Haploinsufficiency A20 (HA20) With Disrupted Domains of A20

Published:2020-09-23 Issue: Volume:11 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Chen Yu,Ye Zhenghao,Chen Liping,Qin Tingting,Seidler Ursula,Tian De'an,Xiao Fang

Funder

National Natural Science Foundation of China

Tongji Medical College, Huazhong University of Science and Technology

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference18 articles.

1. A20 at the crossroads of cell death, inflammation, and autoimmunity;Martens;Cold Spring Harb Perspect Biol.,2020

2. A20 prevents inflammasome-dependent arthritis by inhibiting macrophage necroptosis through its ZnF7 ubiquitin-binding domain;Polykratis;Nat Cell Biol.,2019

3. A20 in inflammation and autoimmunity;Catrysse;Trends Immunol.,2014

4. De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-kappaB signalling;Wertz;Nature.,2004

5. Loss-of-function mutations in TNFAIP3 leading to A20 haploinsufficiency cause an early-onset autoinflammatory disease;Zhou;Nat Genet.,2016

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1. Genetic Mutations Associated With TNFAIP3 (A20) Haploinsufficiency and Their Impact on Inflammatory Diseases;International Journal of Molecular Sciences;2024-07-29

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5. Genomic testing for inborn errors of immunity in seronegative autoimmune hepatitis of childhood;Journal of Hepatology;2024-04

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