STAT3-Mediated Transcriptional Regulation of Osteopontin in STAT3 Loss-of-Function Related Hyper IgE Syndrome-Reference-Cited by-同舟云学术

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STAT3-Mediated Transcriptional Regulation of Osteopontin in STAT3 Loss-of-Function Related Hyper IgE Syndrome

Published:2018-05-17 Issue: Volume:9 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Goel Shubham,Sahu Smrity,Minz Ranjana W.,Singh Surjit,Suri Deepti,Oh Young M.,Rawat Amit,Sehgal Shobha,Saikia Biman

Funder

Indian Council of Medical Research

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference59 articles.

1. STAT 3 mutation in the hyper IgE syndrome;Holland;N Engl J Med,2007

2. Dominant-negative mutations in the DNA-binding domain of STAT 3 cause hyper-IgE syndrome;Minegishi;Nature,2007

3. Autosomal dominant STAT3 deficiency and hyper-IgE syndrome: molecular, cellular, and clinical features from a French national survey;Chandesris;Medicine,2012

4. Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome;Milner;Nature,2008

5. Molecular explanation for the contradiction between systemic TH17 defect & localized bacterial infection in hyper-IgE syndrome;Minegishi;J Exp Med,2009

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