Specific Inhibition of Complement Activation Significantly Ameliorates Autoimmune Blistering Disease in Mice-Reference-Cited by-同舟云学术

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Specific Inhibition of Complement Activation Significantly Ameliorates Autoimmune Blistering Disease in Mice

Published:2018-03-16 Issue: Volume:9 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Mihai Sidonia,Hirose Misa,Wang Yi,Thurman Joshua M.,Holers V. Michael,Morgan B. Paul,Köhl Jörg,Zillikens Detlef,Ludwig Ralf J.,Nimmerjahn Falk

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference45 articles.

1. Pemphigoid diseases;Schmidt;Lancet,2013

2. Emerging treatments for pemphigoid diseases;Ludwig;Trends Mol Med,2013

3. Mechanisms of autoantibody-induced pathology;Ludwig;Front Immunol,2017

4. Granulocyte-derived elastase and gelatinase B are required for dermal-epidermal separation induced by autoantibodies from patients with epidermolysis bullosa acquisita and bullous pemphigoid;Shimanovich;J Pathol,2004

5. Autoantibodies to type VII collagen mediate Fcgamma-dependent neutrophil activation and induce dermal-epidermal separation in cryosections of human skin;Sitaru;Am J Pathol,2002

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1. Advancements in Bullous Pemphigoid Treatment: A Comprehensive Pipeline Update;American Journal of Clinical Dermatology;2023-12-29

2. The role of C5a receptors in autoimmunity;Immunobiology;2023-09

3. Gene Expression Profiling Suggests that Complement Activation Is Important for Blister Formation in Bullous Pemphigoid;Journal of Investigative Dermatology;2023-08

4. Complement: Functions, location and implications;Immunology;2023-05-24

5. The complement receptor C5aR2 regulates neutrophil activation and function contributing to neutrophil-driven epidermolysis bullosa acquisita;Frontiers in Immunology;2023-05-19

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