Activation of Adhesion GPCR EMR2/ADGRE2 Induces Macrophage Differentiation and Inflammatory Responses via Gα16/Akt/MAPK/NF-κB Signaling Pathways-Reference-Cited by-同舟云学术

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Activation of Adhesion GPCR EMR2/ADGRE2 Induces Macrophage Differentiation and Inflammatory Responses via Gα16/Akt/MAPK/NF-κB Signaling Pathways

Published:2017-04-03 Issue: Volume:8 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

I Kuan-Yu,Huang Yi-Shu,Hu Ching-Hsun,Tseng Wen-Yi,Cheng Chia-Hsin,Stacey Martin,Gordon Siamon,Chang Gin-Wen,Lin Hsi-Hsien

Funder

Ministry of Science and Technology, Taiwan

Chang Gung Memorial Hospital, Linkou

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference58 articles.

1. Phagocytosis: an immunobiologic process;Gordon;Immunity,2016

2. From the reticuloendothelial to mononuclear phagocyte system – the unaccounted years;Yona;Front Immunol,2015

3. Macrophage receptors and immune recognition;Taylor;Annu Rev Immunol,2005

4. Human EMR2, a novel EGF-TM7 molecule on chromosome 19p13.1, is closely related to CD97;Lin;Genomics,2000

5. F4/80, a monoclonal antibody directed specifically against the mouse macrophage;Austyn;Eur J Immunol,1981

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2. Adhesion GPCR ADGRE2 Maintains Proteostasis to Promote Progression in Acute Myeloid Leukemia;Cancer Research;2024-04-18

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4. The Posttraumatic Increase in the Adhesion of GPCR EMR2/ADGRE2 to Circulating Neutrophils Is Not Related to Injury Severity;Cells;2023-11-20

5. Biased signalling is structurally encoded as an autoproteolysis event in adhesion G protein‐coupled receptor Latrophilin‐3/ADGRL3;Basic & Clinical Pharmacology & Toxicology;2023-08

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