Wip1 Deficiency Promotes Neutrophil Recruitment to the Infection Site and Improves Sepsis Outcome-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Wip1 Deficiency Promotes Neutrophil Recruitment to the Infection Site and Improves Sepsis Outcome

Published:2017-08-22 Issue: Volume:8 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Shen Xiao-Fei,Zhao Yang,Cao Ke,Guan Wen-Xian,Li Xue,Zhang Qian,Zhao Yong,Ding Yi-Tao,Du Jun-Feng

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference38 articles.

1. Developing a new definition and assessing new clinical criteria for septic shock: for the Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3);Shankar-Hari;JAMA,2016

2. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3);Singer;JAMA,2016

3. Neutrophil dysregulation during sepsis: an overview and update;Shen;J Cell Mol Med,2017

4. Sepsis-induced immune dysfunction: can immune therapies reduce mortality?;Delano;J Clin Invest,2016

5. The pathogenesis of sepsis;Stearns-Kurosawa;Annu Rev Pathol,2011

Cited by 12 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A narrative review of chemokine receptors CXCR1 and CXCR2 and their role in acute respiratory distress syndrome;European Respiratory Review;2024-07

2. Wip1 inhibits neutrophil extracellular traps to promote abscess formation in mice by directly dephosphorylating Coronin-1a;Cellular & Molecular Immunology;2023-06-29

3. Dysregulated neutrophil extracellular traps formation in sepsis;Immunology;2023-06-28

4. Reparixin improves survival in critically ill and transplant patients: A meta‐analysis;European Journal of Clinical Investigation;2023-05-09

5. Mitogen Activated Protein Kinase (MAPK) Activation, p53, and Autophagy Inhibition Characterize the Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) Spike Protein Induced Neurotoxicity;Cureus;2022-12-09

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3