IL-33 and IL-18 in Inflammatory Bowel Disease Etiology and Microbial Interactions-Reference-Cited by-同舟云学术

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IL-33 and IL-18 in Inflammatory Bowel Disease Etiology and Microbial Interactions

Published:2019-05-14 Issue: Volume:10 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Williams Michelle A.,O'Callaghan Amy,Corr Sinéad C.

Funder

Science Foundation Ireland

Crohn's and Colitis Foundation of America

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference57 articles.

1. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines;Schmitz;Immunity.,2005

2. A putative protein of a growth specific cDNA from BALB/c-3T3 cells is highly similar to the extracellular portion of mouse interleukin 1 receptor;Tominaga;FEBS Lett.,1989

3. The innate cytokines IL-25, IL-33, and TSLP cooperate in the induction of type 2 innate lymphoid cell expansion and mucous metaplasia in rhinovirus-infected immature mice;Han;J Immunol.,2017

4. IL-33 improves wound healing through enhanced M2 macrophage polarization in diabetic mice;He;Mol Immunol.,2017

5. T1/ST2 is preferentially expressed on murine Th2 cells, independent of interleukin 4, interleukin 5, and interleukin 10, and important for Th2 effector function;Lohning;Proc Natl Acad Sci USA.,1998

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