SOCS2 Is Critical for the Balancing of Immune Response and Oxidate Stress Protecting Against Acetaminophen-Induced Acute Liver Injury-Reference-Cited by-同舟云学术

SOCS2 Is Critical for the Balancing of Immune Response and Oxidate Stress Protecting Against Acetaminophen-Induced Acute Liver Injury

Published:2019-01-22 Issue: Volume:9 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Monti-Rocha Renata,Cramer Allysson,Gaio Leite Paulo,Antunes Maísa Mota,Pereira Rafaela Vaz Sousa,Barroso Andréia,Queiroz-Junior Celso M.,David Bruna Araújo,Teixeira Mauro Martins,Menezes Gustavo Batista,Machado Fabiana Simão

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference40 articles.

1. Acetaminophen hepatotoxicity;Larson;Clin Liver Dis.,2007

2. Acetaminophen safety and hepatotoxicity—where do we go from here?;Amar;Expert Opin Drug Saf.,2007

3. Acetaminophen hepatotoxicity and repair: the role of sterile inflammation and innate immunity;Jaeschke;Liver Int.,2012

4. Molecular mechanisms of the hepatotoxicity caused by acetaminophen;Nelson;Semin Liver Dis.,1990

5. Inhibition of mitochondrial respiration in vivo is an early event in acetaminophen-induced hepatotoxicity;Donnelly;Arch Toxicol.,1994

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